Hyperosmolar Hyperglycemic State (HHS)
π Definition:
HHS is a life-threatening acute complication of diabetes mellitus, characterized by:
- Severe hyperglycemia
- Marked hyperosmolarity
- Dehydration
- Minimal or absent ketoacidosis
π More common in elderly patients with type 2 diabetes
𧬠Pathophysiology:
Factor |
Effect |
Relative insulin deficiency |
Sufficient to prevent ketogenesis but insufficient to prevent hyperglycemia |
Hyperglycemia |
Osmotic diuresis β profound dehydration |
No significant lipolysis |
Ketone production is minimal or absent |
Plasma hyperosmolality |
CNS dysfunction due to cellular dehydration |
β οΈ Precipitating Factors:
Category |
Examples |
Infection |
Pneumonia, UTI, sepsis |
Drugs |
Thiazides, steroids, antipsychotics |
Non-compliance |
Missed medications |
Stress |
MI, stroke, pancreatitis |
π§ͺ Diagnostic Criteria (ADA):
Parameter |
HHS Values |
Plasma glucose |
>600 mg/dL |
Arterial pH |
>7.30 |
Serum bicarbonate |
>18 mmol/L |
Ketones |
Minimal or absent |
Effective serum osmolality |
>320 mOsm/kg |
Anion gap |
Normal or mildly β |
Mental status |
Altered sensorium common |
π¬ Effective Serum Osmolality:
Effective Osmolality (mOsm/kg)
=2ΓNaβΊ (mEq/L)+Glucose (mg/dL)18
320 mOsm/kg is diagnostic of HHS
π§ Clinical Features:
System |
Manifestations |
General |
Weakness, polyuria, polydipsia |
CNS |
Confusion, lethargy, seizures, coma |
CVS |
Hypotension, tachycardia, signs of hypovolemia |
Renal |
Oliguria due to volume depletion |
π Management of HHS:
β 1. Fluid Resuscitation
- Start with 0.9% NS, 15β20 mL/kg in first hour (~1 L)
- Switch to 0.45% NS based on corrected sodium and osmolality
- When glucose <250 mg/dL β add 5% dextrose
- Total fluid deficit = 8β12 L (more than DKA)
β 2. Insulin Therapy
- Start only after fluids are started
- IV insulin:
- Bolus: 0.1 U/kg (optional)
- Infusion: 0.05β0.1 U/kg/hr
- Goal: β glucose by 50β70 mg/dL/hr
β 3. Electrolyte Correction (esp. KβΊ)
- Serum KβΊ may appear normal or high, but total body KβΊ is depleted
- Replace based on levels:
Serum KβΊ |
Action |
<3.3 mEq/L |
Hold insulin, replace KβΊ first |
3.3β5.3 mEq/L |
Give KβΊ 20β30 mEq/L of fluid |
>5.3 mEq/L |
Monitor without replacing |
β 4. Monitor and Support
- Monitor glucose, electrolytes, osmolality, ABG every 2β4 hrs
- Watch for cerebral edema (especially if glucose drops too fast)
- Start prophylactic LMWH due to hypercoagulable state
π§Ύ Criteria for Resolution of HHS:
Parameter |
Target |
Glucose |
<250 mg/dL |
Mental status |
Improved |
Osmolality |
<300 mOsm/kg |
Vital signs |
Stable |
Electrolytes/ABG |
Normalized |
β οΈ Complications:
Complication |
Risk Factors |
Cerebral edema |
Rapid glucose/osm drop |
Thrombosis |
Hyperviscosity, immobility |
ARDS |
Severe dehydration, infection |
Renal failure |
Prerenal due to volume loss |
π Summary Mnemonic β βHIGH OSMβ for HHS
- H β Hyperglycemia (>600)
- I β Insidious onset
- G β Glucose management
- H β Hypovolemia (fluids!)
- O β Osmolality ββ
- S β Seizures/mental status change
- M β Minimal ketones
π DKA vs HHS: Comparison Table
Feature |
DKA |
HHS |
Underlying Diabetes |
Type 1 (mostly) |
Type 2 (mostly) |
Onset |
Rapid (hours to 1β2 days) |
Insidious (several days to weeks) |
Plasma Glucose |
>250 mg/dL |
>600 mg/dL |
Serum Osmolality |
Mildβmoderate β |
>320 mOsm/kg |
pH |
<7.30 |
>7.30 |
Serum Bicarbonate |
<18 mmol/L |
>18 mmol/L |
Anion Gap |
Elevated (>12) |
Normal or mildly β |
Ketones (urine/serum) |
Present (Ξ²-hydroxybutyrate β) |
Minimal or absent |
Acidosis |
High anion gap metabolic acidosis |
No significant acidosis |
Mental Status |
Alert to comatose (depends on severity) |
Altered sensorium common, seizures possible |
Volume Depletion |
Moderate (~6 L deficit) |
Severe (~8β12 L deficit) |
Typical Age Group |
Younger (children, adolescents) |
Older adults (often >60 years) |
Mortality |
~2β5% |
10β20% (higher) |
Treatment Priorities |
Fluids β Insulin β Electrolytes |
Fluids β Electrolytes β Insulin |
Risk of Cerebral Edema |
Higher in children |
Rare, but possible if glucose β too rapidly |
π‘ Key Differences:
- HHS has much higher glucose and osmolality, but minimal acidosis.
- DKA has marked acidosis and ketosis, but glucose is modestly elevated compared to HHS.